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Publications

Revisiting dopamine-acetylcholine imbalance in Parkinson’s disease: glutamate co-transmission as an exciting partner in crime

CRAGG S. and Zhang Y-F., (2021), Neuron

Axonal Modulation of Striatal Dopamine Release by Local γ-Aminobutyric Acid (GABA) Signalling.

Roberts BM. et al, (2021), Cells, 10

GABA uptake transporters support dopamine release in dorsal striatum with maladaptive downregulation in a parkinsonism model.

Roberts BM. et al, (2020), Nat Commun, 11

CLR01 protects dopaminergic neurons in vitro and in mouse models of Parkinson's disease.

Bengoa-Vergniory N. et al, (2020), Nat Commun, 11

Diabetes Causes Dysfunctional Dopamine Neurotransmission Favoring Nigrostriatal Degeneration in Mice.

Pérez-Taboada I. et al, (2020), Mov Disord, 35, 1636 - 1648

L-type calcium channel contribution to striatal dopamine release is governed by calbindin-D28K, the dopamine transporter, D2-receptors, α2δ-subunits and sex differences

Brimblecombe KR. et al, (2020)

Impairment of Macroautophagy in Dopamine Neurons Has Opposing Effects on Parkinsonian Pathology and Behavior.

Hunn BHM. et al, (2019), Cell Rep, 29, 920 - 931.e7

Plasticity in striatal dopamine release is governed by release-independent depression and the dopamine transporter.

Condon MD. et al, (2019), Nat Commun, 10

Calbindin-D28K Limits Dopamine Release in Ventral but Not Dorsal Striatum by Regulating Ca2+ Availability and Dopamine Transporter Function.

Brimblecombe KR. et al, (2019), ACS Chem Neurosci, 10, 3419 - 3426

Astrocytic striatal GABA transporter activity governs dopamine release and shows maladaptive downregulation in early parkinsonism

Roberts BM. et al, (2019)

Dopamine neuron-derived IGF-1 controls dopamine neuron firing, skill learning, and exploration.

Pristerà A. et al, (2019), Proc Natl Acad Sci U S A, 116, 3817 - 3826

Inhibition of Nigrostriatal Dopamine Release by Striatal GABAA and GABAB Receptors.

Lopes EF. et al, (2019), J Neurosci, 39, 1058 - 1065

Targeted Activation of Cholinergic Interneurons Accounts for the Modulation of Dopamine by Striatal Nicotinic Receptors.

Brimblecombe KR. et al, (2018), eNeuro, 5

Pauses in Cholinergic Interneuron Activity Are Driven by Excitatory Input and Delayed Rectification, with Dopamine Modulation.

Zhang Y-F. et al, (2018), Neuron, 98, 918 - 925.e3

The Striosome and Matrix Compartments of the Striatum: A Path through the Labyrinth from Neurochemistry toward Function.

Brimblecombe KR. and Cragg SJ., (2017), ACS Chem Neurosci, 8, 235 - 242

Pauses in Striatal Cholinergic Interneurons: What is Revealed by Their Common Themes and Variations?

Zhang Y-F. and Cragg SJ., (2017), Front Syst Neurosci, 11

Cortical Control of Striatal Dopamine Transmission via Striatal Cholinergic Interneurons.

Kosillo P. et al, (2016), Cereb Cortex, 26, 4160 - 4169

Striatal dopamine neurotransmission: regulation of release and uptake.

Sulzer D. et al, (2016), Basal Ganglia, 6, 123 - 148

Representation of spontaneous movement by dopaminergic neurons is cell-type selective and disrupted in parkinsonism.

Dodson PD. et al, (2016), Proc Natl Acad Sci U S A, 113, E2180 - E2188

LRRK2 BAC transgenic rats develop progressive, L-DOPA-responsive motor impairment, and deficits in dopamine circuit function.

Sloan M. et al, (2016), Hum Mol Genet, 25, 951 - 963

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