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AIMS/HYPOTHESIS: Hypomagnesaemia (blood Mg2+ <0.7 mmol/l) is a common phenomenon in individuals with type 2 diabetes. However, it remains unknown how a low blood Mg2+ concentration affects lipid and energy metabolism. Therefore, the importance of Mg2+ in obesity and type 2 diabetes has been largely neglected to date. This study aims to determine the effects of hypomagnesaemia on energy homeostasis and lipid metabolism. METHODS: Mice (n = 12/group) were fed either a low-fat diet (LFD) or a high-fat diet (HFD) (10% or 60% of total energy) in combination with a normal- or low-Mg2+ content (0.21% or 0.03% wt/wt) for 17 weeks. Metabolic cages were used to investigate food intake, energy expenditure and respiration. Blood and tissues were taken to study metabolic parameters and mRNA expression profiles, respectively. RESULTS: We show that low dietary Mg2+ intake ameliorates HFD-induced obesity in mice (47.00 ± 1.53 g vs 38.62 ± 1.51 g in mice given a normal Mg2+-HFD and low Mg2+-HFD, respectively, p 

Original publication

DOI

10.1007/s00125-018-4680-5

Type

Journal article

Journal

Diabetologia

Publication Date

09/2018

Volume

61

Pages

2030 - 2042

Keywords

Brown adipose tissue, Energy homeostasis, Hypomagnesaemia, Lipid metabolism, Lipolysis, Magnesium, Obesity, White adipose tissue, β-Adrenergic receptor, 3T3-L1 Cells, Animals, Diet, High-Fat, Insulin Resistance, Lipid Metabolism, Magnesium, Magnesium Deficiency, Male, Mice, Mice, Inbred C57BL, Obesity, Real-Time Polymerase Chain Reaction