Postdoctoral Research Scientist
Dr Haram Park is a neuroscientist whose research focuses on how innate behaviour is expressed and how it is changed within the context of neuropsychiatric diseases. He received his PhD in 2019 at the Korea Advanced Institute of Science and Technology (KAIST) under Prof Eunjoon Kim, where he studied how autism spectrum disorder (ASD)-related behaviours are manifested in different genetic models, as well as how disruption of molecular interactions across the synapse leads to disruptions in innate behaviours. He has presented his findings in numerous international conferences including The Brain Conference, The FENS Forum, and the UK-Korea Neuroscience Symposium.
Haram joined Prof Gero Miesenböck’s lab in DPAG as a Postdoctoral Researcher in 2019 and Magdalen College as a Fellow by Examination in 2020. He has previously held an European Molecular Biology Organization (EMBO) Postdoctoral Fellowship (2020) and currently holds a National Research Foundation (NRF, S. Korea) Postdoctoral Fellowship (since 2021).
Haram’s current project focuses on recent breakthroughs in the Miesenböck lab that revealed how the ‘need to sleep’ is tracked in the brain at a biochemical and electrophysiological level. His aim is to take the findings further to understand the details of sleep homeostasis in the brain.
Splice-dependent trans-synaptic PTP delta-IL1RAPL1 interaction regulates synapse formation and non-REM sleep
Park H. et al, (2020), EMBO JOURNAL
NGL-1/LRRC4C-Mutant Mice Display Hyperactivity and Anxiolytic-Like Behavior Associated With Widespread Suppression of Neuronal Activity
Choi Y. et al, (2019), FRONTIERS IN MOLECULAR NEUROSCIENCE, 12
Shank3 Exons 14-16 Deletion in Glutamatergic Neurons Leads to Social and Repetitive Behavioral Deficits Associated With Increased Cortical Layer 2/3 Neuronal Excitability
Yoo T. et al, (2019), FRONTIERS IN CELLULAR NEUROSCIENCE, 13
NGL-3 in the regulation of brain development, Akt/GSK3b signaling, long-term depression, and locomotive and cognitive behaviors
Lee H. et al, (2019), PLOS BIOLOGY, 17
NGL-1/LRRC4C Deletion Moderately Suppresses Hippocampal Excitatory Synapse Development and Function in an Input-Independent Manner
Cho Y. et al, (2019), FRONTIERS IN MOLECULAR NEUROSCIENCE, 12