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Search results (68)

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Dopamine release from Parkinson’s patient-derived neurons is disrupted due to impaired synaptic vesicle loading

Cramb KML. et al, (2026)

Dynamins maintain nuclear envelope homeostasis and genome stability.

Aveleira C. et al, (2026), Nat Commun, 17

Nucleophagy removes cytotoxic trapped PARP1

Hoslett G. et al, (2025)

Loss of the lysosomal protein CLN3 triggers c-Abl-dependent YAP1 pro-apoptotic signaling.

Domingues N. et al, (2025), EMBO Rep, 26, 6096 - 6120

Intersectin and endophilin condensates prime synaptic vesicles for release site replenishment.

Ogunmowo TH. et al, (2025), Nat Neurosci, 28, 1649 - 1662

MiR-186-5p inhibition restores synaptic transmission and neuronal network activity in a model of chronic stress.

Rodrigues B. et al, (2025), Mol Psychiatry, 30, 1034 - 1046

Role of lipids in interorganelle communication.

Domingues N. et al, (2025), Trends Cell Biol, 35, 46 - 58

TEX264 DRIVES SELECTIVE AUTOPHAGY OF DNA LESIONS TO PROMOTE DNA REPAIR AND CELL SURVIVAL

Lascaux P. et al, (2025), CANCER RESEARCH, 85

TEX264 drives selective autophagy of DNA lesions to promote DNA repair and cell survival.

Lascaux P. et al, (2024), Cell, 187, 5698 - 5718.e26

Connexin43 promotes exocytosis of damaged lysosomes through actin remodelling.

Domingues N. et al, (2024), EMBO J, 43, 3627 - 3649

Upregulation of cholesterol synthesis by lysosomal defects requires a functional mitochondrial respiratory chain.

Agostini F. et al, (2024), J Biol Chem, 300

Loss of the lysosomal protein CLN3 modifies the lipid content of the nuclear envelope leading to DNA damage and activation of YAP1 pro-apoptotic signaling.

Domingues N. et al, (2024)

Up-regulation of cholesterol synthesis by lysosomal defects requires a functional mitochondrial respiratory chain.

Agostini F. et al, (2024), bioRxiv

MiR-186-5p inhibition restores synaptic transmission and neuronal network activity in a model of chronic stress

Carvalho AL. et al, (2023)

Intersectin and Endophilin condensates prime synaptic vesicles for release site replenishment.

Ogunmowo T. et al, (2023), bioRxiv

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