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Search results (11)

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Correction: Mitochondrial impairment, decreased Sirtuin activity and protein acetylation in dorsal root ganglia in Friedreich ataxia models.

Sanz-Alcázar A. et al, (2025), Cell Mol Life Sci, 82

USP30 inhibition improves mitochondrial health through both PINK1-dependent and independent mechanisms

Williamson MG. et al, (2025)

Mitochondrial impairment, decreased sirtuin activity and protein acetylation in dorsal root ganglia in Friedreich Ataxia models.

Sanz-Alcázar A. et al, (2023), Cell Mol Life Sci, 81

Mitochondrial iron and calcium homeostasis in Friedreich ataxia.

Tamarit J. et al, (2021), IUBMB Life, 73, 543 - 553

Calcitriol increases frataxin levels and restores mitochondrial function in cell models of Friedreich Ataxia.

Britti E. et al, (2021), Biochem J, 478, 1 - 20

Calpain-Inhibitors Protect Frataxin-Deficient Dorsal Root Ganglia Neurons from Loss of Mitochondrial Na+/Ca2+ Exchanger, NCLX, and Apoptosis.

Britti E. et al, (2021), Neurochem Res, 46, 108 - 119

PPAR gamma agonist leriglitazone improves frataxin-loss impairments in cellular and animal models of Friedreich Ataxia.

Rodríguez-Pascau L. et al, (2021), Neurobiol Dis, 148

Tau inhibits mitochondrial calcium efflux and makes neurons vulnerable to calcium-induced cell death.

Britti E. et al, (2020), Cell Calcium, 86

Mitochondrial calcium signalling and neurodegenerative diseases.

Britti E. et al, (2018), Neuronal Signal, 2

Frataxin-deficient neurons and mice models of Friedreich ataxia are improved by TAT-MTScs-FXN treatment.

Britti E. et al, (2018), J Cell Mol Med, 22, 834 - 848

Mitochondrial pore opening and loss of Ca2+ exchanger NCLX levels occur after frataxin depletion.

Purroy R. et al, (2018), Biochim Biophys Acta Mol Basis Dis, 1864, 618 - 631