Cookies on this website

We use cookies to ensure that we give you the best experience on our website. If you click 'Accept all cookies' we'll assume that you are happy to receive all cookies and you won't see this message again. If you click 'Reject all non-essential cookies' only necessary cookies providing core functionality such as security, network management, and accessibility will be enabled. Click 'Find out more' for information on how to change your cookie settings.

Search results (11)

« Back to Publications

ctivation of IP3R in atrial cardiomyocytes leads to generation of cytosolic cAMP.

Akerman EC. et al, (2024), Am J Physiol Heart Circ Physiol, 327, H830 - H846

Integrated Proteomics Unveils Nuclear PDE3A2 as a Regulator of Cardiac Myocyte Hypertrophy.

Subramaniam G. et al, (2023), Circ Res, 132, 828 - 848

Phosphodiesterase 2A2 regulates mitochondria clearance through Parkin-dependent mitophagy.

Lobo MJ. et al, (2020), Commun Biol, 3

Multi-Compartment, Early Disruption of cGMP and cAMP Signalling in Cardiac Myocytes from the mdx Model of Duchenne Muscular Dystrophy.

Brescia M. et al, (2020), Int J Mol Sci, 21

Imaging cAMP nanodomains in the heart.

Chao Y-C. et al, (2019), Biochem Soc Trans, 47, 1383 - 1392

Guanylyl cyclase-G is an alarm pheromone receptor in mice.

Chao Y-C. et al, (2018), EMBO J, 37, 39 - 49

Receptor guanylyl cyclase-G is a novel thermosensory protein activated by cool temperatures.

Chao Y-C. et al, (2015), EMBO J, 34, 294 - 306

Guanylate cyclase-G, expressed in the Grueneberg ganglion olfactory subsystem, is activated by bicarbonate.

Chao Y-C. et al, (2010), Biochem J, 432, 267 - 273

Pravastatin attenuates carboplatin-induced cardiotoxicity via inhibition of oxidative stress associated apoptosis.

Cheng C-F. et al, (2008), Apoptosis, 13, 883 - 894

Disruption of guanylyl cyclase-G protects against acute renal injury.

Lin H. et al, (2008), J Am Soc Nephrol, 19, 339 - 348

Peroxisomal proliferator-activated receptor-alpha protects renal tubular cells from doxorubicin-induced apoptosis.

Lin H. et al, (2007), Mol Pharmacol, 72, 1238 - 1245