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Search results (78)

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Distinct roles of the Lyve1 lineage in heart development

Klaourakis K. et al, (2026)

Maternal Anaemia and Congenital Heart Disease in Offspring: A Case-Control Study Using Linked Electronic Health Records in the United Kingdom.

Nair M. et al, (2025), BJOG, 132, 1139 - 1146

Maternal anaemia during pregnancy is associated with an increase in the risk of offspring congenital heart disease: a case-control study using linked electronic health records in England

Nair M. et al, (2024)

Noncoding regulation of epicardial gene expression and epithelial-to-mesenchymal transition during heart development

Vieira JN. et al, (2024), CIRCULATION RESEARCH, 135

Insights into the Role of a Cardiomyopathy-Causing Genetic Variant in ACTN2.

Broadway-Stringer S. et al, (2023), Cells, 12

Better communication between experts is needed to solve the environmental origins of birth defects.

Sparrow DB., (2022), Bioessays, 44

Myhre syndrome is caused by dominant-negative dysregulation of SMAD4 and other co-factors.

Alankarage D. et al, (2022), Differentiation, 128, 1 - 12

Small change, big impact: A Z-disc missense genetic variant causes dramatic morphological changes in the embryonic heart

Jiang H. et al, (2022), JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY, 173, S44 - S44

The onset of circulation triggers a metabolic switch required for endothelial to hematopoietic transition.

Azzoni E. et al, (2021), Cell Rep, 37

Maternal iron deficiency perturbs embryonic cardiovascular development in mice.

Kalisch-Smith JI. et al, (2021), Nat Commun, 12

Maternal iron deficiency perturbs embryonic cardiovascular development in mice

Kalisch-Smith JI. et al, (2021), Nature Communications, 12

Maternal iron deficiency impacts the placental arterial network

Kalisch-Smith JI. et al, (2021)

Functional analysis of a gene-edited mouse model to gain insights into the disease mechanisms of a titin missense variant.

Jiang H. et al, (2021), Basic Res Cardiol, 116

Analysis of Placental Arteriovenous Formation Reveals New Insights Into Embryos With Congenital Heart Defects.

Kalisch-Smith JI. et al, (2021), Front Genet, 12

Heterozygous loss of WBP11 function causes multiple congenital defects in humans and mice.

Martin EMMA. et al, (2020), Hum Mol Genet, 29, 3662 - 3678

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