Search results (15)
« Back to PublicationsCreER activation transiently disrupts angiogenesis by reducing proliferation and promoting apoptosis in vascular endothelial cells
Journal article
Ioannou E. et al, (2026), Angiogenesis, 29
Mutagenesis on a complex mouse genetic background by site-specific nucleases.
Journal article
Davies B. et al, (2024), Transgenic Res, 33, 415 - 426
PHACTR1 modulates vascular compliance but not endothelial function: a translational study.
Journal article
Wood A. et al, (2023), Cardiovasc Res, 119, 599 - 610
Tamoxifen exacerbates morbidity and mortality in male mice receiving medetomidine anaesthesia.
Journal article
Rashbrook VS. et al, (2023), Anim Welf, 32
Cre toxicity in mouse models of cardiovascular physiology and disease.
Journal article
Rashbrook VS. et al, (2022), Nat Cardiovasc Res, 1, 806 - 816
A key role for the novel coronary artery disease gene JCAD in atherosclerosis via shear stress mechanotransduction.
Journal article
Douglas G. et al, (2020), Cardiovasc Res, 116, 1863 - 1874
Tamoxifen-Activated CreERT Impairs Retinal Angiogenesis Independently of Gene Deletion.
Journal article
Brash JT. et al, (2020), Circ Res, 127, 849 - 850
Roles for endothelial cell and macrophage Gch1 and tetrahydrobiopterin in atherosclerosis progression.
Journal article
Douglas G. et al, (2018), Cardiovasc Res, 114, 1385 - 1399
Endothelial Cell Tetrahydrobiopterin Modulates Sensitivity to Ang (Angiotensin) II-Induced Vascular Remodeling, Blood Pressure, and Abdominal Aortic Aneurysm.
Journal article
Chuaiphichai S. et al, (2018), Hypertension, 72, 128 - 138
LOSS OF KIAA1462, A CORONARY ARTERY DISEASE ASSOCIATED GENE, DECREASES ATHEROSCLEROSIS
Conference paper
Douglas G. et al, (2018), CARDIOVASCULAR RESEARCH, 114, S3 - S3
Deficiency in endothelial cell tetrahydrobiopterin increases resistance vascular remodelling, blood pressure, and susceptibility to aortic abdominal aneurysm in response to angiotensin II
Conference paper
Chuaiphichai S. et al, (2018), CARDIOVASCULAR RESEARCH, 114, S90 - S90
Loss of Kiaa1462, a Coronary Artery Disease Associated Gene, Decreases Atherosclerosis
Conference paper
Douglas G. et al, (2018), ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY, 38
Human venous valve disease caused by mutations in FOXC2 and GJC2.
Journal article
Lyons O. et al, (2017), J Exp Med, 214, 2437 - 2452
Human Venous Valve Disease Caused by Mutations in FOXC2 and GJC2
Conference paper
Lyons O. et al, (2017), JOURNAL OF VASCULAR RESEARCH, 54, 62 - 62
Mechanisms underlying human venous valve disease caused by mutations in Foxc2 and connexin47
Conference paper
Lyons OTA. et al, (2017), BRITISH JOURNAL OF SURGERY, 104, 8 - 8

