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Anna Schneider


DPhil Student

I obtained my Medical Degree from Paracelsus Medical University in Salzburg, Austria. There, I first became fascinated with neuroscience when performing dissections and looking at the beautiful anatomy of the brain. In 2017, I was able to spend a research trimester at Harvard Medical School conducting clinical neuroscience research within the Neurosurgery Department of Beth Israel Deaconess Medical Center. During my time there, I first authored three and coauthored two papers and got invited for oral and poster presentations, amongst other to the annual meeting of the New England Neurosurgical Society.

With my team, I studied the development and change of headache perception in patients with unruptured intracranial aneurysms who had undergone treatment with the Pipeline Embolization Device (PED) and defined a Clopidogrel response cut-off value using Light Transmission Aggregometry before PED treatment of unruptured intracranial aneurysms. My clinical research collaboration with Harvard Medical School is still ongoing, and currently, we are investigating the causes of seasonality in neurological diseases, such as migraine and stroke.

I find the brain and central nervous system endlessly intriguing because of its vital role in every aspect of human life and health and I am now applying my fascination with the brain to studying its workings at a molecular and cellular level. The prospect of, by doing so, advancing our understanding of the brain is my motivation to study neuroscience.

I am currently working in the laboratory of Prof. Zoltán Molnár whose research focuses on cerebral cortical development. It seeks to decipher how cerebral cortical neuronal cell fates are determined and how development of cortical functional specialization is governed by genetic and environmental factors. Currently, I am using a conditional knockout mouse model to investigate how blocked vesicular neurotransmitter release in selected projection neurons and hence decreased activity in glutamatergic pyramidal cells influences the number and distribution of GABAergic interneurons in different cortical areas and the striatum.