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In Vivo Post-Cardiac Arrest Myocardial Dysfunction Is Supported by Ca2+/Calmodulin-Dependent Protein Kinase II-Mediated Calcium Long-Term Potentiation and Mitigated by Alda-1, an Agonist of Aldehyde Dehydrogenase Type 2

Woods CE., Shang C., Taghavi F., Downey P., Zalewski A., Rubio GR., Liu J., Homburger JR., Grunwald Z., Qi W., Bollensdorff C., Thanaporn P., Ali A., Riemer RK., Md PK., Mochly-Rosen D., Gerstenfeld E., Large S., Ali ZA., Ashley EA.

Original publication

DOI

10.1161/CIRCULATIONAHA.116.021618

Type

Journal article

Journal

CIRCULATION

Publisher

LIPPINCOTT WILLIAMS & WILKINS

Publication Date

01/09/2016

Volume

134

Pages

961 - +

Keywords

Science & Technology, Life Sciences & Biomedicine, Cardiac & Cardiovascular Systems, Peripheral Vascular Disease, Cardiovascular System & Cardiology, calcium-calmodulin-dependent protein kinase type 2, extracorporeal membrane oxygenation, heart arrest, oxidative stress, PHOSPHOLAMBAN PHOSPHORYLATION, SARCOPLASMIC-RETICULUM, INHIBITION PROTECTS, HEART-FAILURE, CAMKII, CA2+, HYPERTROPHY, LENGTH, CARDIOPULMONARY, RESPONSIVENESS